COPD and Anti Infection Countermeasures: Countermeasures.htm
Lack of certain oil-derived precursor components restricts hormone, and/or enzyme action, and also can lead to immune system up disregulation. Dietary propaganda has classified several essential precursor oils as harmful when they are not.
Mary Enig described some effects of the U.S. diet: low intake of tropical oils causes susceptibilities to microbes, e.g. HIV, RSV and Mycoplasma pneumonia, etc. She also has noted neglected papers that report insufficient butter intake in the presence of high amounts of canola oil (Omega3) has lead to heart lesions. Certain oil molecules are precursor molecules for essential molecules made in the body. Saturated oils have been given a bum rap. Oil intake needs balance, which is missing. (Refs: 7-12)
Restriction or elimination of essential tropical oil precursors in our diet is contributing to gut and respiratory dysbiosis. Lauric acid in coconut oil converts to monolaurin (ML) in the gut. ML dissolves gut microbe’s lipid coatings and interferes with their ability to bind to target cells. (Ref: 3) A surfactant, sodium lauryl sulfate, a common detergent, might play a similar role.
Gut Infections: HIV/AIDS, Measles/ASD, Yersinia entercolitica/Ankylosing Spondylitis Unk/lower back pain.
HIV recovery or reversal has been reported based on coconut oil and/or vinegar, taken incessantly as a drug, multiple times a day. MMR live-vaccine sometimes causes chronic measles gut-infection. Autism (ASD) inflammation might be reduced if this infection is eliminated. Cleaning out the gut, sterilizing with vinegar and water, Rebooting the gut flora and repopulating gut with probiotics, buttermilk, and yogurt should help by revising the gut ecology. Maintain new ecology with daily coconut and palm oils and refresh probiotics with yogurt cultures and live sauerkraut. For IBS, a cabbage fermented culture was found to help. See: www.rejuvenative.com/catalog_one.htm or find similar products in health stores. Buy one use for starter and make your own fermentation with raw cabbage, lightly blended. (Ref 17)
COPD treatment using diet changes, enzymes, and POPG aerosol.
A palm oil derivative, Palmitoyl-oleoyl-phosphatidyl-glycerol (POPG), a phospholipid surfactant, normally found in the lungs, plays a complex beneficial role in binding to respiratory synclinal virus (RSV), to Mycoplasma pneumoniae, and likely to other COPD bacterial components. POPG interferes with the respiratory microbes’ ability to invade epithelial cells, to replicate, and to form plaques/biofilms. It also interferes with RSV’s ability to attach to molecules that stimulate immune reactivity. (Refs: 1,2)
A dietary intake low in palm-derived oils, precursors to POPG, is likely to predispose to COPD, by insufficiently blocking microbial ligand functions.
· Add butter, palm, palm kernel, and coconut oils to the daily diet in amounts of several tablespoons. Use these oils in place of oils from peanut, rapeseed=canola, corn, soybeans, cottonseed, etc. Vinegar taken daily has a similar microbe coat-dissolving effect in the gut. These ingredients can be added appropriately to recipes or taken separately as a supplement.
· POPG is available as a powder in small amounts (100 milligrams) but is expensive. It would be needed in micrograms in an inhaler (~50ug/ml) It could be liquefied and packaged in a spray bottle by a compounding pharmacy.
· The enzyme Serapeptase is known to lyse fibrin, to liquefy mucus, and to destroy respiratory bacterial biofilms. It should be taken as needed to reduce congestion and facilitate breathing.
· Ascorbic acid 2-3 grams every 2 hours 12x per day has controlled serious measles infections (Klenner-13)
· Gut dysbiosis implies a greater need for vitamins: E.g., Vitamin C, B12, A,
· Vitamin A from cod liver oil has reversed ASD for a tunnel-blindness genetically predisposed subpopulation.
· Tetracycline antibiotics: Minocycline, Doxycycline, and OxyTetracycline work against Mycoplasma pneumonia and other atypical pneumonia bacteria.
· See: www.cpnhelp.org/home for help with combined antibiotic protocols (CAP) for persistent Chlamydia pneumonia.
“The properties that determine the anti-infective action of lipids are related to their structure, e.g., monoglycerides, free fatty acids. The monoglycerides are active; diglycerides and triglycerides are inactive. Of the saturated fatty acids, lauric acid(C-12) has greater antiviral activity than caprylic acid (C-8), capric acid (C-10) or myristic acid (C-14). In general, it is reported that the fatty acids and monoglycerides produce their killing/inactivating effect by lysing the plasma membrane lipid bilayer. The antiviral action attributed to monolaurin is that of solubilising the lipids and phospholipids in the envelope of the virus, causing the disintegration of the virus envelope. However, there is evidence from recent studies that one antimicrobial effect in bacteria is related to monolaurin's interference with signal transduction (Projan et al., 1994), and another antimicrobial effect in viruses is due to lauric acid's interference with virus assembly and viral maturation (Hornung et al., 1994).”
Dr Frederick Klenner: (1953) Early Clinical Usage of Vitamin C. Quote:
“Our interest with vitamin C against the virus organism began ten years ago in a modest rural home. Here a patient who was receiving symptomatic treatment for virus pneumonia had suddenly developed cyanosis. He refused hospitalization for supportive oxygen therapy. X-Ray had been considered because of its dubious value and because the nearest department equipped to give such treatment was 69 miles distant. Two grams of vitamin C was given intramuscularly with the hope that the anaerobic condition existing in the tissues would be relieved by the catalytic action of vitamin C acting as a gas transport aid in cellular respiration. This was an old idea; the important factor being that it worked. Within 30 minutes after giving the drug (which was carried in my medical bag for the treatment of diarrhea in children) the characteristic breathing and slate-like color had cleared. Returning six hours later, at eight in the evening, the patient was found sitting over the edge of his bed enjoying a late dinner. Strangely enough his fever was three degrees less than it was at 2 P.M. that same afternoon. This sudden change in the condition of the patient led us to suspect that vitamin C was playing a role of far greater significance than that of a simple respiratory catalyst. A second injection of one gram of vitamin C was administered, by the same route, on this visit and then subsequently at six hour intervals for the next three days. This patient was clinically well after 36 hours of chemotherapy. From this casual observation we have been able to assemble sufficient clinical evidence that prove unequivocally that vitamin C is the antibiotic of choice in the handling of all types of virus diseases. Furthermore it is a major adjuvant in the treatment of at other infectious diseases.
“This experimental “strike” on vitamin C as an antibiotic opened a new avenue of approach to the problem of dealing with the virus bodies. With a great deal of enthusiasm we decided to try its effectiveness with all of the childhood diseases. Measles was singled out more so than the others because of the knowledge that it was a small virus like the one causing poliomyelitis. It was reasonable to assume that if measles could be controlled then Poliomyelitis, too, would have a drug that could prevent as well as cure the disease. The use of vitamin C in measles proved to be medical curiosity. For the first time a virus infection could be handled as if it were a dog on a leash. In the Spring of 1948 measles was running in epidemic proportions in this section of the country. Our first act, then, was to have our own little daughters play with children known to be in the “contagious phase.” When the syndrome of fever redness of the eyes and throat, catarrh, spasmodic bronchial cough and Koplik spots had developed and the children were obviously sick, vitamin C was started.
“In this experiment it was found that 1000mg every four hours, by mouth, would modify the attack. Smaller doses allowed the disease to progress. When 1000mg was given every two hours all evidence of the infection cleared in 48 hours. If the drug was then discontinued for a similar period (48 hours) the above syndrome returned. We observed this of and on picture for thirty days at which time the drug (vitamin C) was given 1000 mg every 2 hours around the clock for four days. This time the picture cleared and did not return. These little girls did not develop the measles rash during the above experiment and although exposed many times since still maintain this “immunity.” Late cases were given the vitamin by needle. The results proved to be even more dramatic. Given by injection the same complete control of the measles syndrome was in evidence a 24 and 36 hour periods, depending entirely on the amount employed and the frequency of the administration. Aborting of these cases before the development of the rash apparently gives no interference to the development of immunity. Recent progress on the rapidity of growth (a development) of the virus bodies by means of the electronic microscope makes intelligent the failure experienced by earlier workers when employing vitamin C on the virus organism (or bodies). Unless the virus is completely destroyed, as demonstrated in the experiments with the virus using measles, the infection will again manifest itself after a short incubation period. Small, single daily doses do not even modify the course of the infection.” The blood half life of vitamin C is 30 minutes with no intake.