Now it is time to question the falsified “oils” statistical arguments
and see how this changes our stroke & heart-disease outcomes. We have been
brainwashed by red herring medicine, biased interpretations, confusing effects
with causes, and aggressive promotion of incomplete/wrong conclusions. See Dr.
Mary Enig: The
Truth About Saturated Fats.
For at least 55 years I have observed the
oils-marketing-generated, pseudo-“health” propaganda & advertising that
polyunsaturated fats are good and saturated fats are bad. That saturated fats
are actually the evil-cause of heart disease. As if you can actually demonize a
molecule. The propaganda says:
·
Red herrings: HDL cholesterol is bad. Later HDL is protective. More recently, HDL is
not protective. HDL levels and HDL/LDL
are measures of a symptom. Studies conflict. Doctors treat their patients in
accord with the latest statistical fad.
·
Non-sequitur:
1) That controlling the cholesterol eaten or the amount made by the body
removes the plaques. One increasingly observed effect of reducing cholesterol
appears to be that of cholesterol starvation, which can adversely affect
cholesterol-using parts of the body more than it does the plaques. (Slowing the
body’s regeneration and causing atrophy of muscles and nerves)
·
Non-sequitur: 2) That saturated fats eaten,
rather than carbohydrates, plays a part in the mechanism of plaque formation.
False, no relationship at all. However some saturated fats are essential to
health and we are not eating enough of them.
·
Non-sequitur: 3) That increasing the HDL/LDL
ratio will reduce the rate of plaque formation. Refuted by recent studies: adding excess Lecithin to the diet
increases the HDL/LDL ratio and should reduce the plaques based on statistical
correlations in other studies. Instead, a chemical mechanism was found where
normal gut bacteria create a rotten fish smelling gas, which is converted in
the liver into a molecule (TMAO) that enhances the growth of the plaques.
Functional biochemistry and bacteriology does not tell us that in-vivo suppressing LDL or HDL cholesterol has much of a negative effect on the bacteria grown plaques. Biofilms may actually influence the HDL/LDL ratio. They certainly control it in the plaque microbial colonies. The marginal difference in heart attack rates if we were to change the serum HDL-C/LDL-C ratio, after the fact, may only be 1 to 2 percent reduction, if any in the heart attack death rate.
Recently it was discovered that lecithin’s component phosphatidylcholine when fed to mice caused their intestinal bacteria to generate a gas that smells like rotten fish. The gas molecules when transferred to the liver are used to make an enzyme, trimethylamine N-oxide or TMAO, molecule. TMAO acts to increase arterial plaques. Thus, Lecithin increases the HDL/LDL ratio to reduce the theoretical risk-factor of heart disease. The HDL/LDL risk factor is positively correlated to plaque formation and elsewhere negatively correlated to heart disease statistics. Which falsifies the prevailing theory.
After enough years to make a difference, heart disease is still with us. We are now redefining the essential HDL levels too low and into an unsafe range. Statins and the way we prescribe them without CoQ10 and the other antioxidant vitamins is now causing measurable increases in nutritional-deficiency-caused harm.
Statistics and Functional Medicine should confirm each
other.
There is a divergence between functional-medicine’s
how-it-works, body-of-evidence and the statistical “conclusions” that drive the
advertising and the dietary compliance engines of oil & fat sales in the
USA and the developed world.
I have observed that when they want to tell you a lie, and sell you an
idea, they often do it with statistics.
This is because it invokes the god of Science. We accept and do not question arguments with
the image of Science. Most people, >99% of them, do not have
the skills and the data needed to question the basis of the statistics-derived
conclusions. The brain just turns
off. Were you aware that the just
mentioned “>99%” was just made up?
See what I mean. So I conclude
that when someone is using statistics, in a public context he might be trying
to sell me something.
Now what to do with the functional-data that belies the
statistics? We just invoke the
minor-devil Anecdotal.
Find any/all exception/s to the statistics, call it/them anecdotal,
and almost all of us will turn away without further questioning.
I am perverse; I have found a lot of insight (and profit) into looking
closely at exceptions to the “rules”.
Many anecdotal reports are well written, detailed, insightful,
and thought provoking. Some even have analysis
and postulate how and why things worked in this particular case. Many come from a person with no particular
axe to grind. Some do come via marketing sites and are still credible. With an anecdotal narrative I can then
postulate new causal relationships and seek out confirmatory proofs.
Most important they can confirm a hunch or give me a new hunch to
confirm.
Most of us do not have the time to look things up. It used to be hard. Now it is easy with the internet. Advertising-driven, paid-for studies &
“research” have created an image of safety for what is sold and have told the
lie, for over 50 years, that natural tropical-oils are harmful. They are not, and some are essential to good
health. By avoiding them, we have
created a lot of unnecessary health problems.
If you Google [coconut ischemic heart
plaques] you find:
www.miracle-of-coconut-oil.com/what-causes-heart-disease.html
Discover the hidden fact about what causes heart disease, and
the simple way to ... In fact, it's called Atheromatous Plaque, the
beginning stage of what causes ... in the blood flow to the brain, you
are more likely to have an ischemic stroke. ...
“Studies indicate that microorganism[s] is[/are] a cause of the injury
on the arterial wall…. When the organism enters into the bloodstream, it will
attack arterial cells until the arterial wall gets damaged….. Blood platelets,
calcium, proteins and other lipids combine together in order to heal the wound
on the arterial wall. This will also cause the plaque sticking to the arterial
wall. In fact, it's called
Atheromatous Plaque, the beginning stage of what causes heart disease….”
The proximate causes of plaques that cause
strokes and heart disease are Chlamydiae pneumoniae and certain other
epithelial invading CWD bacterial forms whose DNA has been found in the
plaques.
texasheart.org/HIC/Topics/HSmart/riskfact.cfm
Major heart disease risk factors for adults are explained. ...
fat include butter fat in milk products, fat from red meat, and tropical oils
such as coconut oil. ... When plaque builds up in
the coronary arteries that supply blood to the heart, you are at ...
[risk.]
“Although we often blame the cholesterol found in
foods that we eat for raising blood cholesterol, the main culprit is the
saturated fat in food. (Be sure to read nutrition labels carefully,
because even though a food does not contain cholesterol it may still have large
amounts of saturated fat.) Foods rich in saturated fat include butter fat in
milk products, fat from red meat, and tropical oils such as coconut oil.”
This:
“main culprit is the
saturated fat in food” is a misleading oversimplification. The unmodified saturated fat, “Lauric acid
increases total cholesterol the most of all fatty acids. But most of the
increase is attributable to an increase in high-density lipoprotein (HDL)
"good" cholesterol. As a result, lauric acid has "a more
favorable effect on Total/HDL cholesterol than any other fatty acid, either
saturated or unsaturated"; a lower Total/HDL cholesterol ratio suggests
a decrease in atherosclerotic risk.”
- Lauric acid
It will be seen that the malnutrition caused by this
“anti saturated fat” propaganda is causing the problem of the
plaques, by depriving us of the foods that would be used by the body to control
the plaque infections. In the New
Guinea, there is an island where the primary fat intake is saturated coconut
oil and there is no heart disease, plaques or strokes, at all, in the native
population that does not eat our [American or European] diet. Go here to read
about this exception
that disproves the rule.
If a risk factor is an “effect” and not a “cause”,
controlling it is useless. Doing so can definitely be harmful. Especially if we
block an enzyme that makes essential nutrients, like we do with statins.
Reducing cholesterol shortens lifetimes and destroys the health of the elderly.
Why do we choose 2, above and
not 1?
Systemic treatment of Chlamydia pneumonia infections (C.pn) is hard, costly, and doesn’t always work. [The same thing with Lyme disease]
We all have Chlamydia pneumonia infections. Ten years ago it was estimated at above 65 % if you were over 50 in age. Now CDC says incidence is unknown. But 2 to 5 million of pneumonia cases each year, so in 50 years the entire population would have had pneumonia at least once.
It is very hard to treat; and it is very persistent. C.pn has multiple CWD forms. It spreads just about everywhere in the body, causing meningo-encephalitis (brain and meninges), myocarditis (heart muscle), and Guillain-Barré syndrome, atherosclerosis, Multiple sclerosis, Chronic fatigue, Asthma, Rheumatoid Arthritis (RA), Fibromyalgia, Chronic sinusitis, Cardiac disease, Interstitial cystitis(bladder), Prostatitis, Alzheimer's disease, Crohn's disease, Inflammatory bowel disease,i etc.
C.pn invades nerves, brain, muscles, kidneys, liver, bladder, prostate, epithelial cells in many tracts---(circulatory, respiratory, gut, urinary), immune cells---(macrophages and monocytes).
The standard single antibiotic courses (two weeks monotherapy) only kill C.pn in one of its three life phases, leaving other live forms of C.pn bacteria which are in other stages to renew infection. This is what creates its persistence.i
What
we have been doing to treat plaques?!
We have been controlling the saturated-fats
“risk-factor” using diet rules. We have been controlling made-cholesterol using
statins, for many years. We have been starving ourselves of essential
cholesterols and causing an unhealthful deficiency in essential molecules, CoQ10
and Heme.
During that time we find more functional medicine
studies proving that certain “abnormal” HDL/LDL ranges are an effect,
not a cause. We also find an increasing number (>15,000) of anecdotal
cases of the harm produced by taking statins. We find the bad statin drug
reactions are being grossly underreported to the government.
We find that many doctors seem brainwashed by the
flood of marketing driven paid for studies of the drug companies, saying
“statins can do no harm”, so they ignore the many patients with statin adverse
reaction symptoms. They do not report the drug reactions.
Our
not treating the cause of sclerotic plaques has huge costs. Controlling HDL/LDL
produces little or no benefit. With no
CoQ10 supplementation, statins produce measurable harm. Finally, we have a
statistical study of statin-produced harm.
Adverse Events of
Statins - An Informal Internet-based Study. We also have studies that show that reducing cholesterol
shortens life. Cited by Dr Malcolm Kendrick: The Great
Cholesterol Myth.
Hypothesis:
Modified (hydrogenated)
oils and oxidized (rancid) fats and oxidized cholesterol are not nutritious,
some can be harmful toxins, or they are warped molecules which do not work in
our biochemistry where natural molecules should fit in normally.
Most of the margarines we use in the US contain
domestic oils that have been hydrogenated to raise their melting point.
We fixate now on the “new bad fats” that are Trans
fats or hydrogenated fats. The problem
is bigger. We are eating junk,
artificial non-food; and not eating the good stuff, natural food, instead. Our
media, driven by commercial interests, are still lying about what is good and
bad.
The fats we do eat are modified, not nutritious, and
possibly harmful. We substitute junk for nutritious natural animal fats, butter
and Natural Tropical Oils. (NTOs)
Unsaturated fats turn rancid rapidly, converting to
more-toxic forms. So we saturate them and convert them to junk fats that at
least do not taste bad. Meanwhile
saturated, stable, nutritious, antimicrobial NTOs could be used naturally
because they are a food we evolved with.
NTOs, if not abused by heat or refining, can provide
natural, fat-soluble, essential vitamins in all their alternate forms. As soon
as we process them with heat they are modified and lose their nutritional
values.
NTOs can be saturated
fats: Lauric, Palmitic, Myristic, Caprylic, Capric acids.
Hypothesis: NTEs are not harmful. They are helpful.
Proofs:
Statins
Block Synthesis of Cholesterol, Ubiquinone, Heme & Other Essential
Molecules:
Block
the Mevalonate Pathway and Get Some Unfortunate Results:
How
To Fix This Malnutrition-Caused Epidemic Of Heart Attacks And Scurvy:
Food
Intake Oils That Are Necessary Or Essential To Health:
According to my definition: Essential Oils
include: Omega 3, Lauric, Palmitic, and
Myristic. Because of their use as steroid hormone-feed stock or as
antimicrobial or immune modulating functions.
The
Sources: Virgin And Cold-Pressed, Unrefined Oils:
Olive, Palm,
Palm Kernel, and/or Coconut oils may have additional vitamin and essential
antioxidant components such as CoQ10, Vitamin
E alternative forms, Vitamin A, etc.
Lauric acid, CoQ10 and AA reverse mitochondrial
dysfunctions by killing infected cells, which are replaced by new cells that
are not infected, because they kill the CWD forms and inhibit the
invasion-helping hyaluronidase enzyme. Appropriate anti CWD and Chlamydia
pneumonia killing antibiotic protocols should be used with the improved
nutrition.
Palmitic acid is a precursor of POPG a surfactant in
the lungs that protects against respiratory syncytial virus (RSV), mycoplasmas,
and other COPD microbe components. It is seen by the chart below that if you
consume exclusively canola oil, you will lack the needed input of palmitic
acid. Butter, high in palmitic acid, was shown essential to prevent health
problems in animals used to test the safety of canola oil. Lard and beef tallow
also are high in palmitic.
Table-
1 Common Food Oils Composition:
See:
http://www.scientificpsychic.com/fitness/fattyacids1.html
http://www.scientificpsychic.com/health/fatty-acids-cholesterol.gif
|
Oil Name |
Lauric C12:0 |
Linoleic C18:2 N:6 w6 |
Alpha Linolenic C18:3 w3 |
Capric C10:0 |
Myristic C14:0 |
Oleic C18:1 |
Palmitic C16:0 |
Palmitoleic C16:1 |
Stearic C18:0 |
Cholesterol |
|
Function |
Anti- Microbes, Immune Support |
Essential Inhibits
T-Lymphocytes |
Essential Fatty
acid Reduces Inflam-mation |
Anti-Microbes, Reduces Inflam-mation |
Snake Oil Cetyl-Myristoleate Reduces Pain |
Human Milk-fat Reduce IL-6 |
Lungs Surfactant Anti- Microbes |
|
Commercial Multi-Use Junk-Fat? |
Brain, Nerves, Hormones, Cell Synthesis, Regeneration |
|
Almond |
|
17% |
- |
|
|
69% |
7% |
|
2% |
|
|
0 |
6-18% |
|
|
0 |
36-80% |
7-32% |
|
1.5% |
|
|
|
50% |
0 |
|
|
20% |
10% |
11% |
|
3.5% |
|
|
|
Butter (Cow) |
3% |
2% |
1% |
|
11% |
29% |
27% |
3% |
12% |
273 |
|
Butter
(Human) |
5% |
9% |
1% |
2% |
8% |
35% |
25% |
3% |
8% |
|
|
39-54% |
1-2% |
- |
3-6% |
15-23% |
4-11% |
6-11% |
|
1-4% |
8% Caprylic C8:0 |
|
|
Canola |
0 |
15% |
10% |
2% |
0 |
32% |
1% |
|
2% |
|
|
Cocoa Butter |
0 |
3% |
- |
- |
0 |
34-36% |
25-30% |
|
31-35% |
|
|
Cod Liver |
- |
5% |
0% |
- |
8% |
22% |
17% |
17% |
- |
|
|
Corn |
0 |
45-56% |
1% |
|
0 |
28-37% |
12-14% |
|
2-3% |
5% C:22
& C:24 |
|
Cottonseed |
0 |
52% |
1% |
|
0 |
18% |
13% |
|
13% |
|
|
Duck |
0 |
11.9% |
.87% |
- |
.7% |
36.7-46% |
21-27% |
- |
3.4-4.7% |
|
|
0 |
14% |
|
|
0.4% |
50% |
21% |
|
9% |
|
|
|
Flax/Linseed |
0 |
7-19% |
53% |
|
0 |
14-39% |
4-9% |
|
2-4% |
|
|
Grape seed |
0 |
58-78% |
|
|
0 |
12-28% |
5-11% |
|
3-6% |
|
|
Hemp |
0 |
57% |
|
|
0 |
12% |
6% |
|
2% |
|
|
Lard |
0 |
6% |
- |
- |
1-2% |
46% |
28% |
3% |
13% |
77 |
|
Lecithin, de-oiled |
0 |
0 |
|
|
0 |
0 |
16% |
|
64% |
~50% Phospholipids |
|
Olive |
0 |
5-15% |
1% |
|
0 |
63-81% |
7-14% |
|
3-5% |
|
|
Palm |
0 |
9-11% |
|
|
0 |
38-40% |
43-45% |
|
4-5% |
|
|
47% |
2% |
|
4% |
14-16% |
18% |
7-9% |
|
3% |
|
|
|
Peanut |
0 |
33% |
|
0 |
0 |
47% |
10% |
|
<10% |
|
|
Safflower |
0 |
70-80% |
|
|
0 |
10-20% |
6-7% |
|
0 |
|
|
Sesame |
0 |
39-47% |
|
|
0 |
37-42% |
8-11% |
|
4-6% |
|
|
Soybean |
0 |
46-53% |
7% |
|
0 |
22-27% |
9-12% |
|
4-6% |
|
|
Sunflower |
0 |
68% |
1% |
|
0 |
16-19% |
7% |
|
4-5% |
|
|
Tallow - Beef |
0 |
2 - 3% |
1% |
- |
6 - 8% |
49 -50% |
27 - 30% |
3% |
14 - 16% |
|
|
Walnut |
- |
51% |
5% |
- |
- |
28% |
11% |
- |
5% |
|
|
Wheat Germ |
0 |
55-60% |
|
|
0 |
13-21% |
13-20% |
|
2% |
|
Anti
Microbial Oils And Derivative Molecules:
The ingested natural oils are converted in the gut
to monoglycerides.
“The properties that determine the anti-infective action of lipids are related to their structure, e.g., monoglycerides, free fatty acids. The monoglycerides are active; diglycerides and triglycerides are inactive [against microbes]. Of the saturated fatty acids, lauric acid(C-12) has greater antiviral activity than caprylic acid (C-8), capric acid (C-10) or myristic acid (C-14).
In general, it is reported that the fatty acids and monoglycerides produce their killing/inactivating effect by lysing the plasma membrane lipid bilayer. The antiviral action attributed to monolaurin is that of solubilizing the lipids and phospholipids in the envelope of the virus, causing the disintegration of the virus envelope.
However, there is evidence from recent studies that one antimicrobial effect in bacteria is related to monolaurin's interference with signal transduction (Projan et al., 1994), and another antimicrobial effect in viruses is due to lauric acid's interference with virus assembly and viral maturation (Hornung et al., 1994).” (Ref-2, below)
“The
residents of Kitava lived exclusively on root vegetables (yam, sweet potato,
taro, tapioca), fruit (banana, papaya, pineapple, mango, guava, water melon,
pumpkin), vegetables, fish and coconuts [27-29]. Less than 0.2% of the caloric
intake came from Western food, such as edible fats, dairy products, sugar,
cereals, and alcohol, compared with roughly 75% in Sweden [30]. The intake of
vitamins, minerals and soluble fibre was therefore very high, while the total
fat consumption was low, about 20 % [28], as was the intake of salt (40-50 mmol
Na/10 MJ compared with 100-250 in Sweden). Due to the high level of coconut
consumption, saturated fat made up an equally large portion of the overall
caloric intake as is the case in Sweden. However, lauric acid was the dominant
dietary saturated fatty acid as opposed to palmitic acid in Sweden.
Malnutrition and famine did not seem to occur.” [Ref-11, below]
The
only deaths from eating coconut oils, was by falling out of a coconut tree
during the gathering process.
Reflections:
It was noted elsewhere in the Kitava island studies that the people were not obese. But they eat a huge amount of natural carbohydrates and fiber. Lauric acid (LA) generates a lot more cholesterol HDL than other fats. LA induced cholesterol is a hormone precursor. Coconuts when fed to cattle increased the energy level and metabolism and the cattle lost fatty weight. This is superficially counterintuitive. Perhaps obesity has a hormonal deficiency component. Insulin is a hormone. Without LA, the anti obesity fat/sugar-metabolism regulatory hormone is missing its fatty precursor, the precursor that coconut oil supplies.
The real complexity of fat/cholesterol/hormone generation pathways at the molecular level is fairly complex. The simplicity of the HDL/LDL model is appealing but is too simple, and mostly useless. We need to catalog each molecule variant shape and see how it might be used. What a lot of work. And for what? A 1-2 percent change in the Heart disease statistics? At the expense of proper cholesterol nutrition.
Proper cholesterol nutrition the body must make cholesterol from the fatty acid input feed stocks.
Simply trying to reduce cholesterols big vs little molecules is bound to have adverse nutritive effects on those body processes that use cholesterols.
So those feedstock molecules that raise cholesterol are probably essential feedstock molecules. These are the tropical oils.
Lets address the real problems:
· Identify the bugs like Lyme’s Bb, and the Bug of all trades Cpn, and others we all have.
· Find all the stages/forms and host cell targets,
· And use a Multi-Multi approach to change our body’s environment
o multi antibiotic targeted to each stage and long enough to work
o Use AA and Benicar to handle the Herx
o Multi nutritional: AA, saturated tropical fats, CoQ10,
o Mitochondrial dysfunction fixer molecules Dehydro AA and others?
o Probiotic gut: best mix of friendly microbes,
o Antimicrobial foods: Ethnic foods are GAS, keep this un-patented, unregulated open to all.
o Viruses and
o Made-viruses with selected genetics: same shapes but without the pathology factors and add an apoptosis countdown-timer to made-virus
References:
Mary Enig’s Papers: Google[enig coconut antiviral]
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