1.
Heart Disease (HD) And Fat
Nutrition: Introduction
2.
Truth about HD and Oils is not
exactly as advertised
3.
HDL/LDL ratio is not the right
measure: Controlling it causes harm
4.
Statistics and Functional Medicine
should confirm each other.
8.
Why choose Red Herring Medicine,
not Functional Medicine?
9.
What we have been doing to treat
plaques?
10.
Hypothesis: Modified oils are bad:
They are.
11.
NTOs are not harmful. Despite the
false dogma, they are helpful. Proofs.
12.
Statins Block Synthesis of
Cholesterol, Ubiquinone (CoQ10), Heme
13.
Scurvy is
the same as AA deficiency
14.
How To Fix This
Malnutrition-Caused Epidemic Of Heart Attacks And Scurvy
15.
Food Intake Oils That Are Necessary
Or Essential To Health Odd and Even chains
16.
Table-1 Common Food Oils Composition
17.
Anti Microbial Oils And Derivative
Molecules
18.
Kitava Island Diet: No heart disease
or strokes from eating exclusively oils from fish and coconuts
19.
Reflections on Kitava Diet
20.
The real problems and some possible
future functional solutions:
21.
References
22.
More
Supporting Details: CoQ10 and Statins
Heart and arterial diseases are a major cause of
death factor. Since medicine uses a
descriptive language, there is a tendency to attribute the described anomalies
as causal factors. In fact they may be
or they may just be symptomatic of a hidden cause or active agent. Stenosis accompanies back pain.
Inflammation caused by infections causes the pain and the
stenosis. Neutralize the infection and the
pain vanishes, but the stenosis remains.
Similarly, with HD, the infection causes inflammation. Scurvy causes ROS proliferation. Oxides attack the epithelial microbes and
also weaken the epithelial surfaces. The plaques both protect the microbes and
the weakened epithelial tissue. Provide
the appropriate antibiotics and lots of Ascorbic acid (vitamin C) and the cause
of the plaques is removed. Proper
nutrition alone can make a big difference. See Pauling’s
atherosclerotic nutrition protocol for HD discussion on our website.
In the following discussion a disproved theory is
said to be falsified. Since our
information flows have gaps and time lags, there is a sizable population component
that is unaware of the falsification.
Sometimes it takes hundreds of years for the consensus to shift, and too
often the falsification event is forgotten several times before it is
appreciated and generally taught.
We are all victims of falsified consensus medicine
as it is practiced in the real world.
Our training on such a complex field of knowledge is incomplete. Because
there is so much information, much of the important discoveries are not taught
or are taught by ignorant persons who get it wrong. John Ely of the University of Washington had some useful things
to say about unprofitable modalities in medicine and nutrition which need to be
practiced, but are not. John
Ely Science of Essential Nutrition
Now it is necessary to question the falsified “oils” statistical
arguments and see how this changes our stroke & heart-disease outcomes. We
have been brainwashed by ‘red herring’ medicine, biased interpretations,
confusing effects with causes, and aggressive promotion of incomplete/wrong
conclusions. See Dr. Mary Enig: The
Truth About Saturated Fats.
For at least 55 years I have observed the
oils-marketing-generated, pseudo-“health” propaganda & advertising that
polyunsaturated fats are good and saturated fats are bad. That saturated fats
are actually the evil-cause of heart disease. As if you can actually demonize a
molecule. The propaganda says:
·
Red herrings: First, HDL cholesterol is bad. Later, HDL is protective. More
recently, HDL is not protective. Truth: HDL levels and HDL/LDL are measures of a symptom. Studies
conflict. Doctors treat their patients in accord with the latest small sample
statistical fad.
·
Non-sequitur:
1) That controlling the cholesterol eaten or the amount made by the body removes
the plaques. One increasingly observed effect
of reducing cholesterol appears
to be that of cholesterol starvation, which can adversely affect
cholesterol-using parts of the body more than it does the plaques. (Slowing the
body’s regeneration and causing atrophy of muscles and nerves)
·
Non-sequitur: 2) That saturated fats, rather
than carbohydrates, play a part in the mechanism of plaque formation. False, no
relationship at all. However some saturated fats are essential to health and we
are not eating enough of them.
·
Non-sequitur: 3) That increasing the HDL/LDL
ratio will reduce the rate of plaque formation. Refuted by recent studies: adding excess Lecithin to the diet
increases the HDL/LDL ratio and should reduce the plaques based on statistical
correlations in other studies. Truth: a chemical mechanism was found where
normal gut bacteria create a rotten fish smelling gas, which is converted in
the liver into a molecule (TMAO) that enhances the growth of the plaques. The
bacteria is a so called probiotic that has both beneficial and harmful features
in its DNA recipes.
HDL/LDL
ratio is not the right measure: Controlling it causes harm.
Functional biochemistry and bacteriology do not tell us that in-vivo suppressing LDL or HDL cholesterol has much of a negative effect on the bacteria grown plaques. The reverse may be true. Microbe filled biofilms may actually influence the HDL/LDL ratio. They certainly control it in the plaque microbial colonies. The marginal difference in heart attack rates if we were to change the serum HDL-C/LDL-C ratio, after the fact, may only be 1 to 2 percent reduction, if any in the heart attack death rate. At the same time other death rate fractions increase by more than this so total death rate is worse.
Many of the HD sudden heart attack deaths may be due to fibrillation which may be aggravated by a dietary or water supply magnesium deficiency. The pain from some chronic infections is reportedly eliminated/reduced for some by Epsom salts baths or poultices.
One of the main causes of atherosclerosis is low AA or scurvy. Oxides proliferate and cause a cascade of toxins to be generated. The pharmacokinetics of AA is not widely understood. The official AA nutritional guidelines are plainly just wrong for persons with chronic infections. In practice, only a few percent of patients is the AA blood level taken; and its high variability is misunderstood. Blood AA is not a measure of AA in the tissues and cells where the scurvy acts in a latent, destructive and toxin-generating manner.
Recently it was discovered that lecithin’s component, phosphatidylcholine, when fed to mice caused their intestinal bacteria to generate a gas that smells like rotten fish. The gas molecules when transferred via the blood to the liver are transformed to make an enzyme, trimethylamine N-oxide or TMAO. TMAO acts to greatly increase arterial plaques’ growth rates. Lecithin at the same time increases the HDL/LDL ratio to reduce the theoretical risk-factor of heart disease. Here, HDL/LDL risk factor is positively correlated to plaque formation and elsewhere negatively correlated to heart disease statistics. This proves that the prevailing theory of beneficial HDL/LDL cholesterol control is false, because it is functionally-controlled by diet and gut microbiome, factors that were not considered in the statistical studies we rely on.
After enough years of statin reduction programs to make a difference, heart disease is still with us, statistics only slightly changed. We are now redefining the limiting HDL levels to such a low level that harmful symptoms and bad drug reactions are emerging. Statins prescriptions and the way we prescribe them without CoQ10 and the other antioxidant vitamins is now causing notable increases in nutrition-deficiency-caused harm. But our harm detection reporting system is also broken, so we do not notice it, except as reported by a few watchdog websites. See our thorough discussion of statin drug caused harm.
Statistics and Functional Medicine should confirm each
other.
There is a divergence between functional-medicine’s
how-it-works, body-of-evidence and the statistical “conclusions” and selected
meta-studies that drive the drug advertising and the nutrition promoting
engines of oil & fat sales in the USA and the developed world. Marketing supported promotions are designed
to influence our consumption and the medical prescription rates.
I have observed that when they want to tell you a lie, and sell you an
idea, they often do it with statistics.
This is because it invokes the god of Science. We accept and do not question arguments with
the image of Science. Most people, >99% of them, do not have
the skills and the data needed to question the basis of the statistics-derived
conclusions. The brain just turns
off. Were you aware that the just
mentioned “>99%” was just made up?
See what I mean. So I conclude
that when someone is using statistics, in a public context he might be
trying to sell me something. I need to
get alternative knowledge and to think more deeply about the matter.
Now what to do with the functional-data that belies the
statistics? Promoters just invoke the
minor-devil Anecdotal.
Find any/all exception/s to the statistics, call it/them anecdotal,
and almost all of us will turn away without further questioning.
Then they also use Evidence-Based methodologies. There is a lot of evidence pro and con. The Con artist will of course select only the
evidence that proves his point and pretend that there is no other
evidence. Who can argue with evidence? The evidence summarized by the Con
artist is slanted; not the whole truth, fraudulent. The suppression of the ideas about
vitamin C in high-level usage, Kalokerinos, Stone, Pauling, Klenner and
Cathcart’s collected works was done in this way. Future studies in directions known to lead to successful undesirable
competition were blocked and their supporting published articles were just
ignored. Incomplete, slanted
Meta-studies were generated and became the citations for these Evidence-Based
frauds. Competing researchers are just
denied funds or approvals, like Thomas McPherson Brown’s proposed antibiotic
long-term protocols proofs. Even after he proved bacteria caused arthritis.
I have found a lot of insight (and sometimes profit) into looking
closely at exceptions to the “rules”.
Some anecdotal reports are well written, detailed,
insightful, and thought provoking. Some even have
analysis and postulate how and why things worked in these particular
cases. Many case history reports come
from a careful observer with no particular axe to grind. Some do come via
marketing sites and may still be credible.
Given an anecdotal narrative I can then postulate new causal
relationships and seek out confirmatory proofs.
Most important, anecdotal reports can confirm a hunch or
give me a new hunch to confirm.
Most of us do not have the time to look things up. It used to be much harder. Now it is easy with searches on the
Internet. It still requires judgment,
but excellence stands out.
Advertising-driven, paid-for studies & planted medical journal
“research” have created an image of safety for what is sold and have told the
lie, for over 50 years, that natural tropical-oils are harmful. They are not, and some saturated fats
provide microbial defenses and are essential building cholesterol, hormone
molecules and to support good nerve, gut and brain health. By avoiding palm and coconut oils, we have
created a lot of unnecessary health problems.
By not eating enough vitamin C (AA) we promote oxides that toxify our
fats and proteins when we suffer from chronic infections.
If you Google [coconut ischemic heart
plaques] you find:
www.miracle-of-coconut-oil.com/what-causes-heart-disease.html
The Truth: Discover the hidden fact about what causes heart disease, and
the simple way to ... In fact, it's called Atheromatous Plaque, the
beginning stage of what causes ... in the blood flow to the brain, you
are more likely to have an ischemic stroke. ...
“Studies indicate that microorganism[s] is[/are] a cause of the injury
on the arterial wall…. When the organism enters into the bloodstream, it will
attack arterial cells until the arterial wall gets damaged….. Blood platelets,
calcium, proteins and other lipids combine together in order to heal the wound
on the arterial wall. This will also cause the plaque sticking to the arterial
wall. In fact, it's called
Atheromatous Plaque, the beginning stage of what causes heart disease….”
The proximate causes of plaques that cause
strokes and heart disease are Chlamydiae pneumoniae and certain other
epithelial invading CWD bacterial forms whose DNA has been found in the
plaques.
texasheart.org/HIC/Topics/HSmart/riskfact.cfm
Misleading:
Major heart disease risk factors for adults are explained. ...
fat include butter fat in milk products, fat from red meat, and tropical oils
such as coconut oil. ... When plaque builds up in
the coronary arteries that supply blood to the heart, you are at ...
[higher risk.]
“Although we often blame the cholesterol found in
foods that we eat for raising blood cholesterol, the main culprit is the
saturated fat in food. (Be sure to read nutrition labels carefully,
because even though a food does not contain cholesterol it may still have large
amounts of saturated fat.) Foods rich in saturated fat include butter fat in
milk products, fat from red meat, and tropical oils such as coconut oil.”
Misleading:
This “main
culprit is the
saturated fat in food” is an oversimplification. The unmodified saturated fat, “Lauric acid
increases total cholesterol the most of all fatty acids. But most of the
increase is attributable to an increase in high-density lipoprotein (HDL)
"good" cholesterol. As a result, lauric acid has "a more
favorable effect on Total/HDL cholesterol than any other fatty acid, either
saturated or unsaturated"; a lower Total/HDL cholesterol ratio suggests
a decrease in atherosclerotic risk.” - Lauric acid
[Another saturated fat, Palmitic acid, is precursor for lung surfactants that alleviate COPD. Palmitic was found to be essential to keep mice alive when fed exclusively highly-promoted canola oil they died.]
More
Truth: It
will be seen that the malnutrition caused by this “anti saturated fat”
propaganda is causing the “problem” of the plaques, by depriving
us of the foods that would be used by the body to control systemic infections
(Yeasts: Caprylic acid), respiratory tract (COPD: Palmitic), gut (HIV: Lauric),
and plaque infections (HD: Lauric).
In
New Guinea, there is an island (Kitava) where the primary fat intake is
saturated coconut oil and there is no heart disease, plaques or strokes, at
all, in the native population that does not eat our [American or European]
diet. Go here to read about this exception
that disproves the rule.
Conclusion: If a risk factor is an
“effect” and not a “cause”, controlling it is useless, perhaps harmful. Doing
so can definitely be harmful if we
block making CoQ10 and HEME, that are essential molecules. Reducing cholesterol
shortens lifetimes and destroys the health and vitality of the elderly. We have seen it happening in ourselves and
for other family members.
Why choose 2-Red Herring
Medicine, not 1-Functional Medicine?
Systemic treatment of persistent Chlamydia pneumonia infections (C.pn) is hard, costly, and doesn’t always work. Relapses happen frequently. The same thing happens with Lyme disease’s persistent Borellia bacteria.
We all have Chlamydia pneumonia infections. Ten years ago CDC estimated it at above 65 % if you were over 50 in age. Now CDC says incidence is “unknown”. But they report 2 to 5 million of pneumonia cases each year, so in 50 years the entire population would have had C.pn pneumonia at least once. The infections are persistent, they do not go away if untreated, and they slowly kill you.
C.pn is very hard to eliminate; it is very persistent. C.pn has multiple CWD forms. It spreads just about everywhere in the body, causing meningo-encephalitis (brain and meninges), myocarditis (heart muscle), and Guillain-Barré syndrome, atherosclerosis, Multiple sclerosis, Chronic fatigue, Asthma, Rheumatoid Arthritis (RA), Fibromyalgia, Chronic sinusitis, Cardiac disease, Interstitial cystitis(bladder), Prostatitis, Alzheimer's disease, Crohn's disease, Inflammatory bowel disease,1 etc.
C.pn invades nerves, brain, muscles, kidneys, liver, bladder, prostate, epithelial cells in many tracts---(circulatory, respiratory, gut, urinary), immune cells---(macrophages and monocytes).
The standard single antibiotic courses (two weeks monotherapy) only kill C.pn in one of its three life phases, leaving other live forms of C.pn bacteria that are in other CWD stages to renew infection. This is what creates its persistence.2 We need a more intelligent protocol methodology. GOOGLE[Trevor Marshall] for an example of a global team trying to develop one. Notable success comes from their great dedication and persistence. Excellence stands out.
What
we have been doing to treat plaques?!
We have been controlling the saturated-fats
“risk-factor” using diet rules. We have been controlling made-cholesterol using
statins, and eaten cholesterol by diet for many years. We have been starving
ourselves of essential cholesterols and causing an unhealthful deficiency in
essential molecules, CoQ10 and HEME.
During that time we find more functional medicine
studies proving that certain “abnormal” HDL/LDL ranges are an effect,
not a cause. We also find an increasing number (>15,000) of anecdotal
cases of the harm produced by taking statins. We find the bad statin drug
reactions are being grossly underreported by practitioners to the government.
We read that some doctors seem brainwashed by the
flood of marketing driven paid for studies of the drug companies, saying
“statins can do no harm”, so they actively ignore the many patients who report
statin adverse reaction symptoms. They do not report the drug reactions.
Our not nutritionally treating the cause of
sclerotic plaques has huge costs. Controlling HDL/LDL produces little or no
benefit. With no CoQ10 supplementation,
statins produce measurable harm. Finally, we have a compelling study of
statin-produced harm. Adverse
Events of Statins - An Informal Internet-based Study. We also have studies that show that reducing cholesterol
shortens life. Cited by Dr Malcolm Kendrick: The Great
Cholesterol Myth.
Hypothesis:
Some modified oils are bad:
Modified (hydrogenated)
oils and oxidized (rancid) fats and oxidized cholesterol are not nutritious,
some can be harmful toxins, or they are warped molecules that do not work in
our biochemistry where natural molecules should fit in normally.
Many of the margarines we use in the US contain
domestic oils that have been hydrogenated to raise their melting point. We
still see dietary propaganda that natural caprylic, palmitic and lauric oils
are harmful in foods when they are not.
We fixate now on the “new bad fats” that are Trans
fats or hydrogenated fats. The problem
is bigger. We are eating modified junk,
artificial non-food non-cheeses; and not eating the good stuff, natural food,
instead. Our media, driven by nationalistic agricultural interests, are still
lying about what is good and bad. It may be good for the balance of payments
but bad for our heath.
The hydrogenated fats we do eat are not nutritious,
and possibly harmful. We substitute junk for nutritious natural animal fats,
butter and Natural Tropical Oils. (NTOs)
Unsaturated fats turn rancid rapidly, converting to
more-toxic forms. So we saturate them and convert them to junk fats that at
least do not taste bad. Meanwhile
saturated, stable, nutritious, antimicrobial NTOs could be used naturally
because they are a food we evolved with.
Natural Tropical Oils See Saturated
Fatty Acids
NTOs are even numbered Carbon chain saturated fats: Lauric, Palmitic, Myristic, Caprylic, Capric, Caproic acids.
Odd numbered Carbon chain saturated fats are subject of Revichi anti microbial protocols. See Revichi Legacy.
NTOs, if not abused by over heating or refining, can
provide natural, fat-soluble, essential vitamins in all their alternate
forms. As soon as we process them with heat they may be modified and may
lose their nutritional values.
Hypothesis: NTOs are not harmful. Despite the false dogma, NTOs are helpful, anti microbials.
Proofs:
Mitochondrial
dysfunction: When a cell is invaded by a microbe, the microbe steals energy from
the cell. Provide more Heme, and CoQ10 and all the cells get more ATP energy.
Your tiredness is lessened. You feel the difference. You feel more alive. Systemic yeast infection causes
mitochondrial energy reduction. Add enough systemic vitamin C (ascorbic acid)
and invaded cells are attacked by the oxidized form of vitamin C (DHA) killing
the invaded cells. See Liposomal
vitamin C for one way to get the high blood concentrations needed so
the AA works like an antibiotic.
Statins: Take statins and the production of CoQ10 and Heme
stops. Cell energy pathways and metabolic
cycles stop working; all cells have metabolic dysfunctions. Fatigue and depression increases. Nerves and
muscles start to atrophy and muscles start to hurt, cells die and are not replaced.
You age faster. (See below) Combine
this with not enough vitamin C: Oxygen transport suffers and too little CoQ10
and too little oxygen degrade heart muscle action, systemic muscle energy
production is reduced. Feelings of increased muscle and mental tiredness are
the result.
Statins
Block Synthesis of Cholesterol, Ubiquinone (CoQ10), Heme & Other Essential
Molecules:
Block
the Mevalonate Pathway and Get Some Unfortunate Results:
Scurvy is the same as AA deficiency. But scurvy is a long time frame dietary malady. There is
also a short term dynamic.
How
To Fix This Malnutrition-Caused Epidemic Of Heart Attacks And Scurvy:
Food
Intake Oils That Are Necessary Or Essential To Health:
According to my definition: “Essential Oils” include: Omega 3, Lauric, Palmitic, Caprylic and
Myristic. Because of their use as steroid hormone-feed stock or as
antimicrobial or immune modulating functions. One might add virgin olive oil
for its anti microbial factors as well as its taste. Goat cheeses are rich in Caprylic
acid (anti yeast) and related factors. Palmitic acid (butter) is a precursor
for a respiratory surfactant (POPG) that is anti-inflammatory.
The
Sources: Virgin And Cold-Pressed, Unrefined Oils:
Olive, Palm,
Palm Kernel, and/or Coconut oils may have additional vitamin and essential
antioxidant components such as CoQ10, Vitamin
E alternative forms, Vitamin A forms, etc.
Lauric acid, Caprylic acid, CoQ10 and AA reverse
mitochondrial dysfunctions by killing infected cells, which are slowly replaced
by new cells that are not infected, because they kill the CWD microbial forms and
inhibit the invasion-helping hyaluronidase penetration enzyme. Appropriate anti
CWD and Chlamydia pneumonia killing antibiotic protocols should be used
with the improved nutrition on a persistent, many months schedule.
Palmitic acid is a precursor of POPG a surfactant in
the lungs that protects against respiratory syncytial virus (RSV), mycoplasmas,
and other COPD microbe components. POPG blocks inflammation-triggering signaling
between microbes and the immune cells that they have learned to invade. It is
seen in the Table 1, below, that if you consume exclusively canola oil, you
will lack the needed input of palmitic acid. Butter, high in palmitic acid, was
shown essential to prevent health problems in animals used to test the safety
of canola oil. Lard and beef tallow also are high in palmitic acid.
Stearic acid is also a saturated fat but has few
reports of antimicrobial functions.
Table-
1 Common Food Oils Composition: for an excellently written overview of fatty acids.
See:
http://www.scientificpsychic.com/fitness/fattyacids1.html
http://www.scientificpsychic.com/health/fatty-acids-cholesterol.gif
|
Oil Name |
Lauric C12:0 |
Linoleic C18:2n-6 w6 |
Alpha Linolenic C18:3n-3 w3 |
Capric C10:0 Caprylic C8:0 Caproic C6:0 |
Myristic C14:0 |
Oleic C18:1 |
Palmitic C16:0 |
Palmitoleic C16:1n-7 |
Stearic C18:0 |
Cholesterol (Per 100g
=3.5 oz.) |
|
Function |
Anti- Microbes, Immune Support |
Essential Inhibits
T-Lymphocytes Inhibits
bacteria protein reductase (FABI) |
Essential Fatty
acid Reduces Inflam-mation |
Anti-Microbes, Reduces Inflam-mation |
Snake Oil Cetyl-Myristoleate Reduces Pain |
Human Milk-fat Reduce IL-6 Inhibits FABI |
Lungs Surfactant Anti- Microbes |
Inhibits FABI |
Commercial Multi-Use Junk-Fat? |
Brain, Nerves, Hormones, Cell Synthesis, Regeneration |
|
Almond |
|
17% |
- |
|
|
69% |
7% |
|
2% |
|
|
0 |
6-18% |
|
|
0 |
36-80% |
7-32% |
|
1.5% |
|
|
|
50% |
0 |
|
|
20% |
10% |
11% |
|
3.5% |
|
|
|
Butter
(Cow) |
3% |
2% |
1% |
|
11% |
29% |
27% |
3% |
12% |
Clarified 200mg |
|
Butter
(Human) |
5% |
9% |
1% |
2% Capric |
8% |
35% |
25% |
3% |
8% |
|
|
39-54% |
1-2% |
- |
3-6% Capric |
15-23% |
4-11% |
6-11% |
|
1-4% |
8% Caprylic C8:0 |
|
|
Canola |
0 |
15% |
10% |
2% Capric |
0 |
32% |
1% |
|
2% |
|
|
|
.7% |
|
|
.3% |
1.3% |
1% |
.3% |
.3% |
77mg |
|
|
Cocoa Butter |
0 |
3% |
- |
- |
0 |
34-36% |
25-30% |
|
31-35% |
|
|
Cod Liver |
- |
5% |
0% |
- |
8% |
22% |
17% |
17% |
- |
|
|
Corn |
0 |
45-56% |
1% |
|
0 |
28-37% |
12-14% |
|
2-3% |
5% C:22
& C:24 3% C:20 |
|
Cottonseed |
0 |
52% |
1% |
|
0 |
18% |
13% |
|
13% |
|
|
Duck |
0 |
11.9% |
.87% |
- |
.7% |
36.7-46% |
21-27% |
- |
3.4-4.7% |
91 mg |
|
0 |
15% |
.9% |
|
0.4% |
47.4% |
22% |
3.5% |
9.6% |
~90 mg?? |
|
|
Flax/Linseed |
0 |
7-19% |
53% |
|
0 |
14-39% |
4-9% |
|
2-4% |
|
|
Goat milk butter, cheese Caprylic
anti yeast |
|
|
|
15% Total 4%Butyric Capric 3% Caproic 3%Caprylic |
|
20-30% |
30% |
|
|
|
|
Grape seed |
0 |
58-78% |
|
|
0 |
12-28% |
5-11% |
|
3-6% |
|
|
Hemp |
0 |
57% |
|
|
0 |
12% |
6% |
|
2% |
|
|
Lard |
0 |
6% |
- |
- |
1-2% |
46% |
28% |
3% |
~13% |
95 mg |
|
Lecithin, de-oiled |
0 |
0 |
|
|
0 |
0 |
16% |
|
64% |
~50% Phospholipids |
|
Neem Margosa |
|
13% |
|
|
|
50% |
18% |
|
14% |
|
|
Olive |
0 |
5-15% |
1% |
|
0 |
63-81% |
7-14% |
|
3-5% |
|
|
Palm |
0 |
9-11% |
|
|
0 |
38-40% |
43-45% |
|
4-5% |
|
|
47% |
2% |
|
4% Capric |
14-16% |
18% |
7-9% |
|
3% |
|
|
|
Peanut |
0 |
33% |
|
0 |
0 |
47% |
10% |
|
<10% |
1.7% Arachidic |
|
Safflower |
0 |
70-80% |
|
|
0 |
10-20% |
6-7% |
|
0 |
|
|
Sesame |
0 |
39-47% |
|
|
0 |
37-42% |
8-11% |
|
4-6% |
|
|
Soybean |
0 |
46-53% |
7% |
|
0 |
22-27% |
9-12% |
|
4-6% |
|
|
Sunflower |
0 |
68% |
1% |
|
0 |
16-19% |
7% |
|
4-5% |
|
|
Tallow - Beef |
0 |
2 - 3% |
1% |
- |
6 - 8% |
49 -50% |
27 - 30% |
3% |
14 - 16% |
95 mg |
|
Walnut |
- |
51% |
5% |
- |
- |
28% |
11% |
- |
5% |
|
|
Wheat Germ |
0 |
55-60% |
|
|
0 |
13-21% |
13-20% |
|
2% |
|
Anti
Microbial Oils And Derivative Molecules:
The ingested natural oils are converted in the gut
to monoglycerides.
“The properties that determine the anti-infective action of lipids are related to their structure, e.g., monoglycerides, free fatty acids. The monoglycerides are active; diglycerides and triglycerides are inactive [against microbes]. Of the saturated fatty acids, lauric acid (C-12) has greater antiviral activity than caprylic acid (C-8), capric acid (C-10) or myristic acid (C-14).
In general, it is reported that the fatty acids and monoglycerides produce their killing/inactivating effect by lysing the plasma membrane lipid bilayer. The antiviral action attributed to monolaurin is that of solubilizing the lipids and phospholipids in the envelope of the virus, causing the disintegration of the virus envelope.
Liposomal vitamin C is lecithin (phospholipids) nano-encapsulated ascorbic acid. It also has an affinity for the microbes’ lipid membranes and the capsule delivers AA to the microbes where it can act as an antibiotic. When the AA is oxidized, it can kill them. DHA is transported inside infected sugar ingesting cells by the same conduits that pass sugars. DHA oxidizes the mitochondria, and induces cell-death (apoptosis).
There is also evidence from recent studies that one antimicrobial effect against bacteria is related to monolaurin's interference with signal transduction (Projan et al., 1994), and another antimicrobial effect in viruses is due to lauric acid's interference with virus assembly and viral maturation (Hornung et al., 1994).” (Ref-2, below)
Palmitic acid (butter) derived POPG also reduces signaling between microbe and immune phagocytes, reducing the opportunity of the microbe to attract a cell to invade as a replication factory. Other drugs also do this but need to be supplemented with antibiotics (tetracyclines) to kill the bacteria forms.
Cholesterol column numbers (above) are grams per 100gm = 3.5 Oz. Standard Sample
“The
residents of Kitava lived exclusively on root vegetables (yam, sweet potato,
taro, tapioca), fruit (banana, papaya, pineapple, mango, guava, water melon,
pumpkin), vegetables, fish and coconuts [27-29]. Less than 0.2% of the caloric
intake came from Western food, such as edible fats, dairy products, sugar,
cereals, and alcohol, compared with roughly 75% in Sweden [30]. The intake of
vitamins, minerals and soluble fiber was therefore very high, while the total
fat consumption was low, about 20 % [28], as was the intake of salt (40-50 mmol
Na/10 MJ compared with 100-250 in Sweden). Due to the high level of coconut
consumption, saturated fat made up an equally large portion of the overall
caloric intake as is the case in Sweden. However, lauric acid was the dominant
dietary saturated fatty acid as opposed to palmitic acid in Sweden.
Malnutrition and famine did not seem to occur.” [Ref-11, below]
The
only deaths from eating coconut oils, was by falling out of a coconut tree
during the coconut gathering process.
It was noted elsewhere in the Kitava island studies that the people were not obese. But they eat a huge amount of natural carbohydrates and fiber. Natural exercise is a factor.
Lauric acid (LA) generates a lot more cholesterol HDL than other fats. LA induced cholesterol is a hormone precursor. Coconuts when fed to cattle increased the energy level and metabolism and the cattle lost fatty weight. This is superficially counterintuitive.
Obesity has a hormonal deficiency component. Insulin is a hormone. Without LA, the anti obesity fat/sugar-metabolism regulatory hormone is missing its fatty precursor, the precursor that coconut oil supplies. Iodine deficiency can lead to adrenal hormone and cortisone cortisol regulation problems. Sea fish diet supplies enough iodine and magnesium and other minerals. Sea salt is helpful compared to regular salt.
The real complexity of fat/cholesterol/hormone generation pathways at the molecular level is fairly complex. The simplicity of the HDL/LDL model is appealing but is too simple, and I believe it is mostly useless.
To really understand how all the relevant molecule pathways work we need an online database, like a Wikipedia page for each molecule. A free access Internet online bio-molecule Beilstein. We need to catalog each bioactive molecule variant shape and see how it might be used and misused by our bacteria invaders. What a lot of work. And for what? Right now with statins HDL/LDL pseudo-science, we are getting only a dubious 1-2 percent shift-change in the Heart disease cause of death outcome statistics. At the expense of proper cholesterol nutrition, we shift “cause of death” to other categories and speeds up aging, and we die sooner of other causes. Negative cost-benefit statistics.
Proper cholesterol nutrition the body must make cholesterol from the fatty acid input feed stocks. Simply trying to reduce cholesterols big vs little molecules is bound to have adverse nutritive effects on those body processes that use cholesterols.
So those feedstock molecules that raise cholesterol are probably essential feedstock molecules. These are the tropical oils.
The real problems and some possible future functional solutions:
· Identify the bugs like Lyme’s Bb, and the “Bug of all trades”, Cpn, and others (mycoplasmas) we all have.
· Find all the stages/forms and host cell targets; microbiology basic research is incomplete or un-cataloged.
· Find and document the antibiotic and antimicrobial food susceptibilities of the various forms of each pathogenic invader.
· CRC has data on microbes, listing what biochemicals they manufacture; Gene sequencing correlates to this; link these data bases and fill in the gaps of knowledge. Put it online with low cost access to practitioners and researchers.
· Use a Multi-Multi approach to change our body’s environment, See our discussions of the Ketonic Protocol.
o Multi antibiotic targeted to each microbe form and stage and strong&long enough to work
o Multi microbe treatments: Yeasts, Viruses, Mycoplasmas, Protozoa, CWD forms, Bacteria, Spirochetes, Worms, etc
o Use AA and Benicar to handle the Herx toxin flares of microbe die-off
o Multi nutritional: AA, saturated tropical fats, CoQ10, Caprylic for yeast, minerals: Ca, Mg, etc
o Mitochondrial dysfunction fixer molecules Dehydro AA and others: Glutathione, etc?
o Antimicrobial foods: Ethnic foods are generally accepted as safe (GAS), keep this un-patented, unregulated open to all.
· Future and Far out Solutions:
o Probiotic gut: best mix of friendly microbes, after cataloging their manufactured-molecule genetics
o Gene engineered Probiotics: with great care, after we know what we are doing, make deft improvements to Probiotic microbes to make more good molecules and eliminate the bad molecules and effects.
o Viruses and Made-viruses with designer genetics: Same immune generating outer shapes but without the pathology factors and add multiple apoptosis countdown-timers to made-viruses.
o We can do computer directed design to manufacture for silicon. Do the same with microbes made to order with multiple layered safety factors to control rogue proliferation.
Mary Enig’s Papers: Google[enig coconut antiviral]
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By KFP Updated June
1012
Copyright 2012 KF and KM Poehlmann
