Personal experience has proved repeatedly that a few grams of AA will relieve coughing urgency within a few minutes. Where continued coughing causes a chain reaction of irritation, stimulated by environmental causes, the AA works to relieve the itch that stimulates the cough. The “cure” is temporary, more AA is needed to continue the relief. Eventually the high need for AA diminishes. COPD exacerbations may be soothed by Liposomal AA and sodium ascorbate solution in a nebulizer. The most extreme itchy cough is from Whooping Cough, caused by Pertussis infection.
A GreenMedInfo paper by Dr. S. Humphries cites AA as a treatment for Pertussis (Whooping Cough). AA, in high enough multi gram dosages, is an effective acute neutralizer of all the bacterial toxins generated by the infections that the DPT vaccine protects against. With AA powder so effective against viruses and bacteria (in 1-3 hour, repeated, several-gram oral AA dosages, 24/7 for about a week), many may question the medical industry’s overstated need for the whooping cough (Bordetella. pertussis bacteria) vaccine, which actually is reported not to work very well. IV Sodium ascorbate can achieve even higher antiviral effects.
The Original Omerod Canadian Paper showed low dosages of AA worked to speed up recovery from whooping cough. The AA intake was at least 1/10th of an effective sustained medicinal dose: experimental dosage was too little and not frequent enough to effect a rapid cure. But benefits from even these low AA dosages were remarkable.
A 1937 Ormerod Follow-up Paper confirms that 350 – 100 mg/day amounts of AA temporarily reduces symptoms. It did not go far enough: larger (1-3 gram) amounts of AA, administered every 1 to 3 hours, would have shown even greater benefits. Remember AA’s blood half lifetime is ½ hour. So the Ormerod dosages and lack of frequency must have left the patients with low (unmeasured) AA antioxidant blood levels for much of each day, allowing the bacteria to proliferate.
AA is an essential food-element that we do not make, but need systemically to sustain life and good health. There is no practical upper limit to its safety. Its blood half lifetime is 30 minutes. When we are sick we convert antioxidant AA from a helpful form to a dangerous form (dehydroxy AA) that is rapidly excreted. In the presence of toxins or allergies, we can easily run out of helpful antioxidant vitamin C (AA).
Vitamin C’s effectiveness remains “controversial” because knowledge of its effectiveness, its low cost, its wide availability and its safety threaten so many other toxic patented (high profit) medicines that sustain the “ethical” drug industry. It may make many of them unnecessary.
Nonetheless, AA is still highly useful as complementary “medicine” to neutralize life-threatening endotoxins released by dying bacteria and by invaded body cells that have been killed by prescription antibiotics and oxides. The toxins and low systemic antioxidant AA blood levels and high concentrations of oxidized AA (DHA) induce an allergic like histamine flare reaction. Several grams of AA intake will reduce or eliminate the severity of drug-induced toxemia and allergic flares. Intake of AA must be repeated frequently, because it reacts and is consumed, unlike a vitamin that may be persistent for days.
AA reduces or prevents drug and vaccine adverse reactions that produce toxins, oxides and inflammation. Illness or antibiotic reactions cause systemic or local toxemia. Frequently repeated AA replenishment is needed to keep up with the relentless generation of toxins. AA intake must continue until all the microbes are eliminated and all toxins are flushed from the body. This may take several days to weeks, of elevated, frequent AA intake.
How much Vitamin C. AA intake, in amounts 50 times larger than the (~60 milligram) RDA, i.e., several grams per 1-3 hours, when periodically administered, provides toxin-neutralizing relief. Symptoms do abate within a few minutes.
Repeated intake of enough AA provides relief from adverse drug reactions, from anaphylaxis-like toxic shock symptoms, from smoke inhalation and carbon monoxide poisoning, from snake and insect venoms, from food poisoning toxins, from jellyfish stings and sunburn, from plant poisons (mushroom and mold mycotoxins and poison ivy), from alcohols and barbiturate intoxication, and from allergic reactions of every kind.
Dehydroascorbic acid (DHA) is the oxidized (and oxidizing) form of AA. DHA is produced instantly when AA meets a toxin molecule and acts to neutralize it. DHA, an oxidant, is toxin-generating in its actions.
An important critical measurement of vitality is the AA/DHA ratio in the blood. The AA/DHA = Antioxidant/Oxidant concentrations of ascorbic acid. If AA/DHA is much greater than one (~7) , one is in good health. If AA/DHA is near or less than 1 and decreasing, the patient is in a death spiral, characterized in the extreme by symptoms of anaphylaxis. Measuring this dynamic ratio may be hard to do. But the symptoms of anaphylaxis have been reversed so many times by the immediate administration of 6-25 of grams of IV sodium ascorbate (AA), which will tip the AA/DHA ratio’s balance back to the safe range.
This effect does not last long, and more IV or oral AA needs to be administered repeatedly for a long time until the patient is past the time when the toxins (endotoxins, exotoxins or allergens) have stopped being generated and all are neutralized. This may take days to weeks. Sometimes the patient recovers normal vitality in a few minutes, and if no more AA is administered, within hours or days, a rapid severe relapse can result.
Anaphylaxis is a severe, life threatening, systemic allergic reaction to a toxin. AA blood levels plummet and systemic histamine levels rise exponentially. AA/DHA ratio is near or less than one and decreasing as the allergens cause a toxin release cascade. Bradykenin is one name associated with the toxin of anaphylaxis.
Pre-loading systemic AA via low nutritional doses reduces the chance and severity of anaphylaxis and allergies. In crisis, rapid high systemic administration of adrenaline causes the release of up to about 6 grams of AA stored in the adrenal glands, reversing anaphylaxis and severe systemic allergies, stopping the generation of histamine from mast cells. The AA released neutralizes the Bradykenin toxin and others that are formed in the presence of microbes or allergens. Raising AA blood levels may be only temporary unless additional supplemental AA is administered.
Because shortage of AA and surplus of DHA are the primary causal factors of anaphylaxis, the administration of tens of grams of IV sodium ascorbate may be preferable to use of adrenaline alone, which has but a one-time and limited effect. Use of both, together, may be optimal, since repeated intake of AA by oral, parenteral injection and IV methods can protect against the effects of continuously generated/released toxins of every kind. See References 2,3, and 4, below.
Intensive care medicine needs to take heed of the relationship between antioxidant AA and DHA blood concentrations. When antioxidant AA is depleted and DHA predominates, then many of the biochemical pathways of the body stop working. Systemic release of histamine occurs and blood levels of histamine rise exponentially. The eventual result is death.
It is time for medical doctrine to recognize the need to control the AA/DHA ratio to keep it in the safe range. Patient outcomes will necessarily improve in critical cases if AA/DHA is high. AA (sodium ascorbate) administration in tens of grams per hour can easily do this. It now is time to discard all anti-AA prejudices in medical teaching, doctrine, and practices. AA pharmacokinetics and pharmaco dynamics should be taught. AA biochemistry is highly dynamic and interacts rapidly with vital chemical pathways.
Routine emergency administration of IV AA is as important as maintaining proper electrolyte balances. A lot of mortality and morbidity will be avoided if AA is used as a first aid toxin neutralizer.
It should be recognized that treating AA as a limited intake vitamin, and restricting its intake to one dose per day can lead to swift and extreme AA depletion. The amount of the water-soluble AA depletion can be calculated using the ½ hour blood half lifetime. In case of continuously generated toxins, sudden death may result for cases of high oxidizing stress, toxemia and anaphylaxis, if frequent AA intake is not provided.
Liposomal AA (L-AA) can provide 5 times better gut-to-blood transfer compared to water soluble AA. Its blood retention is longer. It provides higher blood-to-brain transport efficiencies. L-AA targets microbe and infected body cells having lipid capsules and it delivers the AA with 7 to 10 times greater clinical effectiveness compared with IV sodium ascorbate on an AA gram-for-gram basis.
KFP Copyright 2012 by KF & KM Poehlmann, all rights reserved.
Revised September, 2014